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Organic compounds that contain a carbon atom bonded to a halogen atom, and an oxygen atom via a double bond; commonly derived from an oxoacid by replacing a hydroxyl group with a halogen atom.
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N-Phthaloyl-β-alanine (hapten FP) is an FX-type hapten that does not contain a CH2S moiety. It can be coupled to OVA using the mixed anhydride method and exhibits more heterologies.
FX-type hapten.
Does not contain a CH2S moiety.
Can be coupled to OVA using the mixed anhydride method.
Exhibits more heterologies.
Purity of 99.18%.
Molecular formula: C11H9NO4.
Soluble in DMSO (≥ 100 mg/mL).
Store powder at -20°C for 3 years or 4°C for 2 years.
Store in solvent at -80°C for 6 months or -20°C for 1 month.
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N-(Chloroacetyl)-2-methoxybenzamide is a chemical compound primarily used as a drug intermediate for the synthesis of various active compounds. It is intended for research use only.
Used as a drug intermediate
Utilized for the synthesis of various active compounds
Intended for research use only
Not sold to patients
Not available for sale in certain territories as a controlled substance
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Palmitoyl Tetrapeptide-3 (Palmitoyl Tetrapeptide-7) is a synthetic peptide corresponding to amino acids 341-344 of the human immunoglobulin heavy chain. It stimulates phagocytosis, reduces interleukin-6 (IL-6) secretion in keratinocytes, and inhibits the inflammatory response of skin to UVB radiation. This peptide also possesses anti-inflammatory and anti-aging effects, reducing skin wrinkles by promoting the production of elastic fibers in the papillary dermis.
Stimulates phagocytosis.
Reduces interleukin-6 (IL-6) secretion in keratinocytes.
Inhibits the UVB radiation-exposure inflammatory response of skin.
Provides anti-inflammatory and anti-aging effects.
Reduces skin wrinkles by promoting elastic fiber production.
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1-Methylnicotinamide (1-MNA 3-Carbamoyl-1-methylpyridin-1-ium Trigonellamide) chloride is an active endogenous metabolite of nicotinamide that exhibits anti-inflammatory and anti-thrombotic activities 1-Methylnicotinamide enhances tumor vasculature formation and markedly increases prostacyclin (PGI2) generation
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